Based on the higher order structure of actin microfilaments characterizing different areas of an interphase cell, which area would you expect to contain a high concentration of Arp2/3 proteins present?

Piloting and revising the questionnaire or interview schedule is a step that must be included when building a new measure.

A junction that connects adjacent cells directly to one another through catenin proteins and depends on high Calcium ion concentration to form is called:

Which cell’s mitochondria do you expect to contain more cristae?

Qualitative research is performed to enable discovery when there is insufficient prior understanding of a topic, or when preconceived concepts and predefined tools have inadequately captured the phenomena of interest.

TRUE or FALSE: The stability of an actin microfilament depends on the presence of a “GTP cap”

Prior to the 20th century, many sailors suffered from scurvy, which we now know is caused by vitamin C deficiency. Which types of cell junctions would you expect a scurvy patient to experience a loss of? (Hint: Think about what they adhere the cell to)

In brown fat cells, high expression of an uncoupling protein in mitochondria allows protons (H+’s) to flow back across the inner mitochondrial membrane in the direction that is energetically favorable for them to flow, rather than passing through an ATP synthase. High expression of an uncoupling protein is favorable for hibernating animals, because the proton (H+) motive force generates the following (instead of ATP):

Tropomyosin protein is:

Which of the following does NOT occur when nitrous oxide (N2O) is administered in a dentist’s office? (Note: Nitrous oxide is converted to nitric oxide (NO)).  A figure from lecture and a description of the NO signaling pathway is included below to help. (Note: This is a “reading/lecture comprehension” question, and not a question that tests your memorization skills. It is important to me that students understand the dynamic nature of signaling pathways — how they can be regulated and disrupted. I am not trying to trick you!)     Description of pathway: Acetylcholine released from a nerve terminal bind to a G-protein-coupled receptor on an endothelial cell of a blood vessel. The receptor then activates a G protein (not pictured), which stimulates IP3 synthesis and release of calcium ions, which activates nitric oxide synthase (NOS). NOS causes endothelial cells to produce nitric oxide from the amino acid arginine. Because nitric oxide is small and hydrophyllic, it can just diffuse across the membrane of the endothelial cell that it is produced in and INTO the neighboring smooth muscle cells where it acts as a signal.  Once inside the smooth muscle cell, nitric oxide binds to and activates guanylyl cyclase, which then stimulates the synthesis of cyclic GMP (cGMP) from GTP. The cyclic GMP triggers a response that causes the smooth muscle cells to relax, increasing blood flow through the vessel.