The hippocampus is involved in:

Which of the following correlation coefficients expresses the weakest degree of relationship between two variables?

Discrete distributions are sometimes used as approximations of continuous distributions:

Which is the fastest, easiest, and least intrusive means of obtaining a breast biopsy?

Which nuclear medicine imaging agent(s) can localize in breast cancer lesions?

Data collected from approximately the same period of time from a cross-section of a population are called:

PET breast imaging can detect which of the following:

Current trends show that breast cancer death rates are declining in the US.  Which of the follow reasons attributes to the decline?

3. How does ischemic cell death cause a fatal response to myocardial infarction? Relatedly, how can the type I Interferon response be targeted to provide cardioprotection post-myocardial infarction? A. How does release of self-DNA trigger innate immune response upon ischemic cell death? B. How does the IL-1 pathway contribute to post-MI inflammation? C. Describe how IL-1 blockade improves clinical outcomes of heart diseases? D. How does IRF3 and IFNAR deficiency protect against MI?

2. Identify and describe what you know about the different origins of inflammation that can contribute to development of cardiovascular disease? A. Describe how inflammation plays a role in plaque calcification in vascular smooth muscle. B. Briefly describe different risk factors that increase inflammation and affect the cardiovascular system. C. Describe the immune system activators and effectors that contribute to myocardial inflammation, e.g. non-cellular and cellular components, complement, T cell and B cell responses and other immune cells. D. Which cytokines are typically considered pro-inflammatory or anti-inflammatory? E. Describe how dyslipidemia contributes to inflammatory risk and atherosclerosis. F. Describe interventions with anti-inflammatory effects that improve cardiovascular disease. G. Describe how IRF7 acts as a negative regulator of cardiac hypertrophy and its potential as a therapeutic target to counter pathological cardiac hypertrophy. H. How does the IFN-gamma/Stat5 axis protect pressure overload-induced cardiac hypertrophy?